Sympathetic premotor neurons in the rostral ventrolateral medulla (RVLM) are intrinsically
sensitive to hypoxia and are responsible for the sympathoexcitation evoked by acute
cerebral ischaemia. Here we consider two mediators of hypoxia sensitivity in this
population: the oxygen-sensing molecule HO-2, and non-neuronal ATP release.
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Article info
Publication history
Received:
May 15,
2013
Identification
Copyright
© 2013 Published by Elsevier Inc.
