Sympathetic premotor neurons in the rostral ventrolateral medulla (RVLM) are intrinsically sensitive to hypoxia and are responsible for the sympathoexcitation evoked by acute cerebral ischaemia. Here we consider two mediators of hypoxia sensitivity in this population: the oxygen-sensing molecule HO-2, and non-neuronal ATP release.
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Received: May 15, 2013
© 2013 Published by Elsevier Inc.